The Progression
The earliest record of the progress in my CKD was the doctors noticed in 2002 that my creatinine levels had risen and was 1.3 (normal < 1.0). This was during the investigation to have a large blood clot removed from my hip. One thought is that the large clot, also known as a pseudotomor, which was the size of a football in 2003, could have put pressure on the kidney and exacerbated the CDK. Nobody knows. The doctors were hesitant to do a kidney biopsy for fear of causing an internal bleed. I saw a nephologist, Dr. Roger Rodby (RR), at RUSH, Chicago for the first time in 2005. He was referred to me by my hematologist then, Dr. Valentino. Dr. RR continued my traetment to maintain my blood pressure with Enalapril. He believed that Enalapril was kidney-protective. Also, to help my kidney he also added hydroclorothiazide and started me on a low dose combo pill- Enalapril-HcTz (HcTz is a diuretic). His hypothesis on the CKD was that either it was caused by prolonged untreated hypertension of due to the hepatitis B exposure. I was exposed to Hep B due to blood transfusions to treat the hemophilia in India. I also saw a hepatologist, Dr. Shah, at RUSH who investigations (2011) ruled out Hep B as a cause for the CKD, Meanwhile, my blood pressure, did not remain under control and first Dr. RR tried increasing the Enalapril-HcTz. To this he added a new medicine- Toprol, a beta blocker. This worked, however, it made me very drowsy during the day while at work, Toprol also caused my pulse to drop significantly to 50s. After a month (2011)with this side effect Toprol was dropped and Amlodipine was added. Amplodipine is calcium-channel blocker. This again was not effective beyond a year and the hunt was on to find a drug combination that work on my b.p.
The earliest record of the progress in my CKD was the doctors noticed in 2002 that my creatinine levels had risen and was 1.3 (normal < 1.0). This was during the investigation to have a large blood clot removed from my hip. One thought is that the large clot, also known as a pseudotomor, which was the size of a football in 2003, could have put pressure on the kidney and exacerbated the CDK. Nobody knows. The doctors were hesitant to do a kidney biopsy for fear of causing an internal bleed. I saw a nephologist, Dr. Roger Rodby (RR), at RUSH, Chicago for the first time in 2005. He was referred to me by my hematologist then, Dr. Valentino. Dr. RR continued my traetment to maintain my blood pressure with Enalapril. He believed that Enalapril was kidney-protective. Also, to help my kidney he also added hydroclorothiazide and started me on a low dose combo pill- Enalapril-HcTz (HcTz is a diuretic). His hypothesis on the CKD was that either it was caused by prolonged untreated hypertension of due to the hepatitis B exposure. I was exposed to Hep B due to blood transfusions to treat the hemophilia in India. I also saw a hepatologist, Dr. Shah, at RUSH who investigations (2011) ruled out Hep B as a cause for the CKD, Meanwhile, my blood pressure, did not remain under control and first Dr. RR tried increasing the Enalapril-HcTz. To this he added a new medicine- Toprol, a beta blocker. This worked, however, it made me very drowsy during the day while at work, Toprol also caused my pulse to drop significantly to 50s. After a month (2011)with this side effect Toprol was dropped and Amlodipine was added. Amplodipine is calcium-channel blocker. This again was not effective beyond a year and the hunt was on to find a drug combination that work on my b.p.
No comments:
Post a Comment